Myocarditis is more common than Commotio Cardis

When doctors make a diagnosis, several things are considered. How often does that condition happen? In what situation or age group does that illness affect?

Last night on Monday Night Football, millions of NFL fans saw something familiar to soccer fans for the first time. A player who went into cardiac arrest.

Damar Hamlin, a Buffalo Bills player, collapsed and went into cardiac arrest after he tackled the Cincinnati Bengals wide receiver Tee Higgins on the field.

BREAKING — Tonight on NFL’s Monday Night Football, Buffalo Bills Safety Damar Hamlin inexplicably collapses on field. Medical Crews administer CPR.

Prayers for him, his family, and his teammates. pic.twitter.com/WtEfex02zL

— The HighWire (@HighWireTalk) January 3, 2023

Cardiopulmonary resuscitation and chest compressions were applied. A defibrillator was used and shocked his heart. Damar was immediately brought to the University of Cincinnati Medical Center by ambulance.

Presently, he is on life support and listed in critical condition. Some cardiologists believe that the cause of cardiac arrest is commotio cardis.

What is Commotio Cardis?

Commotio cardis a rare cause of sudden death. It is due to a low-energy trauma to the chest wall.

Stat Pearls describes the etiology of Commotio cordis[3]

Commotio cordis most commonly results from an impact to the left chest with a hardball (e.g., a baseball) during sports activity.

The sudden focal distortion of the myocardium results in ventricular fibrillation, causing sudden cardiac arrest in an otherwise structurally normal heart.

It is distinct from a traumatic injury to the heart, like cardiac contusion or rupture or penetrating chest injuries.

It occurs predominantly in children and adolescents 5 to 15 without preexisting heart disease. It has been reported in ice hockey, lacrosse, softball, and after fistfights.[1]

Notice that American football is not included. Why? It could be that the athletes wear protective equipment, which spreads the impact to a wider surface area.

Source: By Willyp15 – Own work, CC BY-SA 4.0, https://commons.wikimedia.org/w/index.php?curid=58998196

Furthermore, in the history of American football, after millions of high-speed tackles by superb athletes weighing more than 200 lbs, there has been no report of Commotio cardis.

In Commotio Cordis, ventricular fibrillation can occur if the chest trauma occurs at a specific time in the cardiac cycle, specifically if the impact occurs within the window from 30 to 15 msec before the peak of the T wave on the electrocardiogram.[1]

That was the finding in an experimental model of Commotio Cordis in animals.[1]

The figure below shows the vulnerable period in the cardiac cycle when Commotio Cordis can occur in mild blunt chest trauma.

Source: By Agateller. from Wikipedia. Public Domain, https://commons.wikimedia.org/w/index.php?curid=12596910

Myocarditis?

Another possibility is ventricular fibrillation due to myocarditis. The National Institute of Health and the Centers for Disease Control recognize myocarditis after mRNA vaccination as a clinical entity.

I do not know if Hamlin is vaccinated, but the NFL has strict COVID vaccination rules.

USA Today reported in November 2021 that according to the rules approved by the National Football League and NFL Players Association, unvaccinated players are subject to more stringent regulations than vaccinated players.

Unvaccinated players must wear masks “at all times when inside the Club facility,” including while working out in the weight room. They must have daily PCR testing. And they are prohibited from coming within six feet of other unvaccinated players while traveling or eating meals.

Those rules can coerce players to get the jabs so they can keep their place on the roster.

In an Associated Press report, the NFL said that 95% of the players had been vaccinated.

If it is vaccine-induced myocarditis, why does it manifest more in males and during physical activity?

Dr. Flaviano Cadegiani, one of the physicians at the Front Line Covid-19 Critical Care Alliance, FLCCC, may have an answer.

Based on his review of the medical journals about the epidemiology, autopsy, molecular, and physiological studies, he hypothesized that catecholamines are a crucial trigger of COVID-19 mRNA-induced myocarditis.[2]

Catecholamines are the hormone adrenaline and noradrenaline. They are responsible for the fight-or-flight responses. Here are his reasons for his conclusion.

SARS-CoV-2 mRNA vaccine-induced myocarditis primarily affected young males, while the risk was not observed following COVID-19 infection

Independent autopsies or biopsies of patients who presented post-SARS-CoV-2 mRNA vaccine myocarditis in different geographical regions enabled the conclusion that a primary hypercatecholaminergic state was the key trigger of these events

SARS-CoV-2 mRNA was densely present, and SARS-CoV-2 spike protein was progressively produced in adrenal medulla chromaffin cells, which are responsible for catecholamine production

The dihydroxyphenylalanine decarboxylase enzyme that converts dopamine into noradrenaline was overexpressed in the presence of SARS-CoV-2 mRNA, leading to enhanced noradrenaline activity

Catecholamine responses were physiologically higher in young adults and males than in other populations

Catecholamine responses and resting catecholamine production were higher in male athletes than in non-athletes

Catecholamine responses to stress and its sensitivity were enhanced in the presence of androgens

Catecholamine expressions in young male athletes were already high at baseline, were higher following vaccination, and were higher than those in non-vaccinated athletes.

Dr. Cadegiani’s hypothesis explains why myocarditis is common among COVID-vaccinated athletes – More than 50% of vaccinated athletes with Myocarditis.

COVID shots cause more arrythmias than other vaccines, which is why the word myocarditis is now part of the vocabulary.

According to a study by Shi et al., arrhythmias can happen after getting any COVID jabs. I discussed their paper on Cardiac Arrhythmias after COVID injections. Part 2

One up-and-coming Japanese baseball player died from fulminant myocarditis. A professional athlete who died of fulminant myocarditis after the Moderna jab.

Myocarditis is more common than usually reported. The only prospective study that followed young students after COVID vaccination showed – A whopping 1,660 myocarditis cases per 100K COVID injections.

Incidences of Commotio Cordis and Myocarditis

According to a recently updated Stat Pearls on Commotio Cordis, less than 30 cases of Commotio Cordis are reported annually.[3] If they say vaccine-induced myocarditis is rare, Commotio cordis is much rarer!

In contrast, there are 2,878 reported myocarditis after the COVID jabs in VAERS. Note that VAERS is underreported by a factor of 100, based on the Lazarus report. Therefore, myocarditis should be around 287,800

Source: VAERS [4]

And there are 164 cases of ventricular fibrillation (VF). Make that 16,400 if multiplied by 100.

Source: VAERS [5]

We should all pray for Damar Hamlin for his recovery. His big advantage is that he is physically fit and strong, which makes his chances much better.

Someone close to him should talk about his vaccine status to stop speculations. If he got the shots, the NFL should change its discriminatory policy on COVID-19.

Side note: One of the Bills’ players is Cole Beasley, a wide receiver. He refused the shot and was released during the off-season. The reason was supposed to make room for the salary cap.

A few weeks ago, he was rehired by the Bills. It makes you wonder what his teammates are saying to him now.

An important sequel to this is Anoxic Brain Injury.

Don’t Get Sick!

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Related:

References:

Link, M et al. An Experimental Model of Sudden Death Due to Low-Energy Chest-Wall Impact (Commotio Cordis). N Engl J Med 1998; 338:1805-1811.
DOI: 10.1056/NEJM199806183382504
Cadegiani FA. Catecholamines Are the Key Trigger of COVID-19 mRNA Vaccine-Induced Myocarditis: A Compelling Hypothesis Supported by Epidemiological, Anatomopathological, Molecular, and Physiological Findings. Cureus. 2022 Aug 11;14(8):e27883. doi: 10.7759/cureus.27883. PMID: 35971401; PMCID: PMC9372380.
Tainter CR, Hughes PG. Commotio Cordis. [Updated 2022 Sep 19]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2022 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK526014/
United States Department of Health and Human Services (DHHS), Public Health Service (PHS), Centers for Disease Control (CDC) / Food and Drug Administration (FDA), Vaccine Adverse Event Reporting System (VAERS) 1990 – 12/23/2022, CDC WONDER On-line Database. Accessed at http://wonder.cdc.gov/vaers.html on Jan 3, 2023 12:55:29 PM
United States Department of Health and Human Services (DHHS), Public Health Service (PHS), Centers for Disease Control (CDC) / Food and Drug Administration (FDA), Vaccine Adverse Event Reporting System (VAERS) 1990 – 12/23/2022, CDC WONDER On-line Database. Accessed at http://wonder.cdc.gov/vaers.html on Jan 3, 2023 8:43:23 AM

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