This article talks about two studies that shed light on why patients with severe COVID-19 and those who died post-vaccination have extensive blood clots in their system.
Yesterday I posted an article from Natural News about the blood clots obtained from autopsied people who passed away after the COVID-19 vaccination. One unusual finding of the blood clots is that they are tough, fibrous, and “rubbery.”
Blood clots happen after physical or microscopic injuries from infections and inflammation of blood vessels. After a few days, the body dissolves the blood clot after the damage is repaired.
The spontaneous dissolution of blood clots is essential because, in daily living, blood clots form in response to injuries to the blood vessels. If blood clots are not dissolved, arteries will be permanently blocked, leading to organ failure or limb loss.
The peer-reviewed article, SARS-CoV-2 spike protein S1 induces fibrin(ogen) resistant to fibrinolysis: implications for microclot formation in COVID-19, explains why blood clots rapidly form in COVID-19 patients and those who had COVID-19 vaccination.[1]
In their experiment, the S1 protein of SARS-CoV-2 was added to the platelet-poor plasma. Platelet-poor plasma (PPP) has a small number of platelets. PPP was used to minimize the effects of platelets on clotting. That way, only the impact of the S1 protein on clotting is measured.
The S1 protein is a part of the spike protein, which is made by the body after mRNA (Pfizer and Moderna) and DNA (AstraZeneca and Johnson and Johnson) COVID-19 injections.
Results
The study showed that adding S1 protein promotes rapid clot formation in four ways.
1. Increase platelet activation
Platelets need to be activated to become sticky to one another before clot formation starts. The images below (C, D) show hyperactivated platelets clumping together after adding S1 protein. These platelets are ready to clot!
2. Fibrin with amyloid formed
The images below (E-H) show the formation of spontaneous and anomalous fibrin(ogen) deposits with an amyloid nature after adding the spike protein.
Fibrin is typically formed on blood clots and serves as a scaffold. Images (E – F) show the spontaneous and anomalous fibrin deposits after adding spike protein.
Images (G, H) display the abnormal fibrin deposits that are amyloid in nature (arrows) after adding spike protein.
Amyloids are not part of the clotting process; thus, their presence here is abnormal. Aspirin, blood thinners, and clot busters used in hospitals cannot dissolve amyloid. That explains why many patients have amputated limbs when they get a clot.
Other scientists have also shown the formation of amyloid in the presence of SARS-CoV-2 spike protein. I discussed that at – The SARS-CoV-2 spike protein can form amyloids seen in lung, blood, and nervous system disorders
3. Sticky blood clots
The next results show that the clot that forms with the addition of spike protein is more adherent to the sides of a tube that mimics a blood vessel.
In healthy plasma (A) (without COVID-19 or spike protein), clot formation was a relatively slow and gradual process, resulting in the formation of a modest clot (arrow). Clots formed in plasma without spike proteins were small and limited to the flow channel’s walls.
In contrast, after adding spike protein, the clots are larger and more adherent to the wall. Images (C-D) are clots from the plasma of patients with COVID-19.
(E-F) shows healthy plasma clots with the addition of spike protein. It is bigger than the clot in (A) immediately above. That is what blood clots look like after COVID shots.
An added observation when the authors were cleaning the tube is that the tube from the plasma without the spike protein added was easy to clean n with high-speed water flow.
However, there was a complete failure to dislodge or disturb COVID-19 clots from the channels.
To summarize, adding spike protein to the blood results in platelet activation and a more significant formation of fibrin with amyloid characteristics.
All these lead to the development of an abnormal blood clot that is difficult to dislodge.
4. Clots are harder to dissolve
The other study by the same authors [2] found that various inflammatory molecules are higher in the blood of those with acute COVID-19 and Long COVID/PASC.
These inflammatory molecules irritate the inner lining of the blood vessels and stimulate clot formation.
Of particular interest was a substantial increase in α 2-antiplasmin, which inactivates plasmin. Plasmin dissolves blood clots. Antiplasmin inactivates plasmin.
To make it short, the net result of the increase in α 2-antiplasmin is a blood clot that persists longer.
The presence of inflammatory markers after SARS-CoV-2 infection is also reproduced in another study that I discussed at:
Inflammatory molecules found in Long COVID patients
Relevance
The research explains the poor prognosis of those with severe COVID-19 patients and the autopsy findings of patients who died after their COVID injections.
Deceased COVID-19 patients
Autopsied patients are noted to have widespread blood clots. Those findings are described in case series from New York, New Orleans, and internationally.
The results from New Orleans prompted the treatment guidelines to start blood thinners for COVID-19.
Blood clots in the vaccinated who died
A series of three and seventeen autopsies in Italy showed thromboembolism. In a series of 18 post-mortems in Germany, all had cardiovascular findings like heart attacks and strokes, but only four were causally related to the vaccines.
In Norway, four autopsied patients had VITT or Vaccine-Induced Thrombocytopenic Purpura. VITT is a new clotting disorder defined only after the start of the COVID vaccination.
Many people with a pre-existing cardiovascular disease will have the cause of death declared unrelated to the shots, like those with diabetes, coronary artery disease, and hypertension.
However, we can argue that the tendency of the spike protein formed after the shots promote further clot formation that can progress to a heart attack or stroke.
Long COVID Syndrome
Diseases are not black or white — they are shades of gray. In between those who are healthy and those who die are people who are in-between and long-suffering.
Long COVID syndrome is also known as long-haul COVID, post-acute COVID-19, post-acute sequelae of SARS CoV-2 infection (PASC), long-term effects of COVID, and chronic COVID.
Long COVID is the persistence of COVID symptoms for four weeks after infection.
Most of the symptoms of Long COVID are due to a lack or decrease in blood and oxygen supply to the internal organs.
- Tiredness or fatigue that interferes with daily life
- Symptoms that get worse after physical or mental effort (also known as “post-exertional malaise”)
- Difficulty breathing or shortness of breath
- Chest pain
- Fast-beating or pounding heart (also known as heart palpitations)
- Difficulty thinking or concentrating (sometimes referred to as “brain fog”)
- Headache
- Sleep problems
- Dizziness when you stand up (lightheadedness)
- Pins-and-needles feelings
- Stomach pain
- Joint or muscle pain
- Changes in menstrual cycles
There is hope.
The Front Line Critical Care Alliance has a treatment overview on managing Long COVID syndrome. You can read that at
The I-RECOVER Management Protocol for Long Haul COVID-19 Syndrome
The FLCCC also has a treatment protocol for Post COVID Vaccine Syndrome
I presented it at The I-RECOVER Post-Vaccine Treatment Protocol
Arteries and veins are not just tubes. My article, The Magical Endothelium, describes the excellent work of a blood vessel to maintain its patency and deliver oxygen throughout the body.
Truth heals. Lies kill. Don’t Get Sick!
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Related:
- Retinal complications after COVID shots
- RNA splice study shows why AstraZeneca and Janssen jabs are clot shots
- Blood Vessel Damaging Proteins of the SARS-CoV-2
- Cerebral Thrombosis after the Pfizer Covid-19 Vaccine
- The High Risk of Deadly Brain Clots in the J & J COVID Vaccine
- This study shows a Ten-Fold Risk of Developing Blood Clots after the COVID Vaccines.
- You got the COVID shot and found that others developed blood clots. Now what?
- Platelet Changes Cause Blood Clots in COVID-19
- Unidentified Foreign Bodies in the Vaccines Form Clots
- Know the Signs of Vaccine-Induced Blood Clotting
References:
- Grobbelaar LM et al. SARS-CoV-2 spike protein S1 induces fibrin(ogen) resistant to fibrinolysis: implications for microclot formation in COVID-19. Biosci Rep. 2021;41(8):BSR20210611. doi:10.1042/BSR20210611
- Pretorius, E., Vlok, M., Venter, C. et al. Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 (PASC) is accompanied by increased levels of antiplasmin. Cardiovasc Diabetol 20, 172 (2021). https://doi.org/10.1186/s12933-021-01359-7
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