This article was updated with English, Spanish and Mandarin audio on November 21, 2025
🎧 Introducción en audio — Español (Latinoamérica)
En este audio comentaremos un caso clínico donde una persona con síntomas de parkinsonismo experimentó mejoría después de usar nattokinasa. Revisaremos qué ocurrió, cómo se interpretan estos hallazgos y por qué este tipo de reportes deben analizarse con cautela. Esta información es educativa y no reemplaza el consejo médico profesional.
音频简介（中文）：
大家好，欢迎收听今天的节目。
这一集我们将分享一个特别的案例：一位长期出现帕金森样症状的病人，在使用纳豆激酶后，身体状况出现了明显改善。节目中会介绍相关机制、最新研究，以及这位病人的转变。内容仅供健康教育参考，希望能带给您新的思考与启发。
About the Author
As a physician with decades of experience in internal medicine and emergency medicine, I have evaluated many patients with neurological, vascular, and inflammatory conditions. This article summarizes scientific literature, case observations, and emerging hypotheses related to neurodegeneration and nattokinase. It is written for educational purposes to help readers understand what current evidence does—and does not—show.
Recently, I published Prostate and Vascular problems relieved with Nattokinase. In it, I described a case of a man in his mid-60s whose symptoms of long-standing prostate pain and vascular problems in the legs improved after one month of a nattokinase and serratiopeptidase combination.
His doctors are at a loss on what is happening with him because all the tests they ran were unremarkable. In passing, I mentioned that he had developed a blunt facial affect, shuffling steps, and resting tremors.
All of them are Parkinsonian symptoms because they are characteristic of Parkinson’s disease. He was never diagnosed, but I have suspected it for months.
Understanding the Significance of This Case
Parkinsonian symptoms—such as shuffling gait, tremor, and facial masking—can arise from many causes, including neurodegenerative disease, inflammation, infection, metabolic dysfunction, or medication effects.
This article discusses one case in which symptoms improved after supplementation. While intriguing, single cases cannot establish causation. Instead, they serve as early signals that may warrant further scientific investigation.
The image below shows the typical posture of someone with Parkinson’s disease.
Interpreting Research on Spike Protein and Prions
Some of the studies and reviews discussed here—including those by Seneff, McCullough, and others—present hypotheses about the spike protein, prions, and neurodegenerative processes.
These papers are part of an ongoing scientific debate.
- Some findings come from laboratory studies, preprints, narrative reviews, or animal models.
- Others are not universally accepted and require more replication.
To maintain scientific accuracy, readers should understand that these hypotheses are emerging, not definitive.
Today I read the narrative review A Potential Role of the Spike Protein in Neurodegenerative Diseases. Two of the authors are familiar to most people: Stephanie Seneff and Peter McCullough.
They described the different mechanisms of how SARS-CoV-2 can induce brain degeneration by producing prions in the brain. They also found that the COVID vaccines, which contain the spike protein, can also produce prion proteins but to a much greater degree.
Prions are misfolded proteins that can cause “mad cow” disease and Creutzfeldt-Jacob Disease (CJD) in humans.
The scary part about prions is that they are transmissible. That is why the beef industry uses enzymes that can degrade prions in the bone meal for cattle.
Side note: In beef processing, the bones not sold are ground up and fed to the cattle for calcium and proteins. If one cow has bovine spongiform encephalopathy or mad cow disease and is fed back to the rest of the herd, they can all have mad cow disease.
SARS-CoV-2, Spike Protein, and Prions
Going back to CJD, Jean Claude Perez, Claire Moret-Chalmin, and Luc Montagnier (RIP), the Nobel Prize-winning virologist, wrote Towards the emergence of a new form of the neurodegenerative Creutzfeldt-Jakob disease: Twenty-six cases of CJD declared a few days after a COVID-19 “vaccine” Jab.
It is a report of 26 people who developed CJD very soon after they received their first or second dose of Pfizer, Moderna, and AstraZeneca “vaccines.”
Of these 26 cases, 20 had died at the time of writing this article while 6 were still alive.
The 20 deaths occurred only 4.76 months after the injection. Among them, 8 of them lead to a sudden death (2.5 months).
All this confirms the radically different nature of this new form of CJD, whereas the classic form requires several decades.
Case reports described in preprints—such as the 26 CJD cases—do not establish a proven causal relationship between vaccination and prion disease. These reports represent temporal associations and must be interpreted with caution until supported by large-scale epidemiologic studies.
Established surveillance systems have not confirmed a causal link. However, discussing early observations is useful for generating scientific questions that can be formally studied.
Another research detected alpha-synuclein proteins and inflammation in the brain of monkeys after a SARS-CoV-2 infection. The alpha-synuclein protein accumulates in the brains of people with Parkinson’s disease.
A preprint study showed Lewy bodies developing in the brain of two macaque monkeys after a SARS-C0V-2 infection. Lewy bodies are also seen in Parkinson’s disease and Lewy-Body Dementia.
In short, several studies link SARS-CoV-2 and the COVID jabs to brain deterioration.
Yesterday, I released Nattokinase Degrades Amyloids and Prions. The article discusses a study showing that nattokinase can degrade amyloids and prions better than the enzymes they use for the bone meal to destroy “mad cow” prions.
Safety and Appropriate Use of Nattokinase
Nattokinase is a proteolytic enzyme with fibrinolytic and anticoagulant effects.
- People taking warfarin, DOACs, aspirin, or other anticoagulants should avoid nattokinase unless supervised by a clinician.
- Those with bleeding disorders, recent surgery, ulcers, or uncontrolled hypertension should consult a physician before use.
Supplements that influence clotting are biologically active and should be used carefully—especially in individuals with neurological symptoms.
Nattokinase is one of the best supplements out there. I’m not exaggerating if I say it should be in the water supply. Forget about fluoride. Nattokinase also strengthens the teeth by depositing calcium in them.
Nattokinase has anti-coagulant, anti-hypertensive, clot-dissolving, lipid, and blood sugar-lowering properties. It can even diminish atherosclerosis. The links are below.
Nattokinase is also a protease or protein enzyme. It can destroy the SARS-CoV-2 spike protein. Two papers have shown that.
- Nattokinase Degrades the SARS-CoV-2 Spike Protein
- Another Study shows Nattokinase can Destroy the S1 Spike Protein
Anyway, our guy who is the subject of Prostate and Vascular problems relieved with Nattokinase sounded happy right before I wrote this article. He was more lively, and the monotone in his voice was gone. His appetite has returned.
I now recall that he was more lively and had more facial expressions recently when I saw him. His appetite has improved.
The improvement in Parkinsonian symptoms observed in this case may have been influenced by many factors, including reduced inflammation, better sleep, improved vascular function, placebo effect, or natural fluctuation of symptoms.
Although nattokinase has shown biochemical effects on fibrin, blood flow, and amyloid breakdown, no clinical trials currently show that nattokinase treats Parkinson’s disease.
This case offers an interesting hypothesis, not a proven therapy.
To conclude, it looks like nattokinase solved not only his prostate and vascular problem but also his Parkinsonian symptoms.
Many cases of neurodegenerative diseases are reported in VAERS – Parkinsonism resolved by Nattokinase
Medical Disclaimer
This article is for informational and educational purposes only. It does not diagnose, treat, or prevent disease. Research on spike protein, prions, and neurodegeneration is evolving and includes early-stage studies and hypotheses.
Nattokinase should not replace medical evaluation for Parkinsonism or any neurological condition. Anyone with tremors, gait changes, facial masking, or cognitive symptoms should seek prompt evaluation from a qualified healthcare professional.
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